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  Indian J Med Microbiol
 

Figure 2: The proposed mechanism of cardiovascular manifestations of COVID-19. Hyperinflammatory state secondary to cytokine storm, increased angiotensin II, low ACE2 levels and APLA contribute to thrombus formation in coronary and pulmonary vasculature. APLA, antiphospholipid antibodies; IL, interleukin; IFN-γ, interferon-gamma; MCP, monocyte chemoattractant protein; TNF-α, tumour necrosis factor-alpha. Source: Refs 24, 25.

Figure 2: The proposed mechanism of cardiovascular manifestations of COVID-19. Hyperinflammatory state secondary to cytokine storm, increased angiotensin II, low ACE2 levels and APLA contribute to thrombus formation in coronary and pulmonary vasculature. APLA, antiphospholipid antibodies; IL, interleukin; IFN-γ, interferon-gamma; MCP, monocyte chemoattractant protein; TNF-α, tumour necrosis factor-alpha. <i>Source</i>: Refs 24, 25.