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CLINICAL IMAGE
Year : 2017  |  Volume : 145  |  Issue : 1  |  Page : 147

Fitz-Hugh-Curtis syndrome


Department of Gastroenterology, Bombay Hospital & Institute of Medical Sciences, Mumbai 400 020, Maharashtra, India

Date of Submission05-Sep-2015
Date of Web Publication30-May-2017

Correspondence Address:
Pravin M Rathi
Department of Gastroenterology, Bombay Hospital & Institute of Medical Sciences, Mumbai 400 020, Maharashtra
India
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Source of Support: None, Conflict of Interest: None


DOI: 10.4103/ijmr.IJMR_1417_15

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How to cite this article:
Sonavane AD, Rathi PM. Fitz-Hugh-Curtis syndrome. Indian J Med Res 2017;145:147

How to cite this URL:
Sonavane AD, Rathi PM. Fitz-Hugh-Curtis syndrome. Indian J Med Res [serial online] 2017 [cited 2020 Feb 22];145:147. Available from: http://www.ijmr.org.in/text.asp?2017/145/1/147/207226

A 23 yr old woman presented to the department of Gastroenterology at Bombay Hospital, Mumbai, India, in July 2015 with an acute onset, sharp right hypochondriac pain; intermittent fever and gradually progressive symmetrical abdominal distension for the preceding two weeks. On physical examination, ascites was evident. On analysis, the ascitic fluid was culture-negative and exudative with normal adenosine deaminase value. C-reactive protein was elevated. Her blood culture and test for human immunodeficiency virus were negative. A computerized tomogram of the abdomen was performed [Figure 1]. She underwent an exploratory laparoscopy that revealed flimsy adhesions between the inter-bowel loops [Figure 2], between the abdominal wall and bowel loops and along the liver surface, ascites and features suggestive of perihepatitis and right salpingitis. Based on these findings, the diagnosis of Fitz-Hugh-Curtis syndrome was made. An endocervical swab did not reveal any pathological organisms as the patient by then had received seven days of intravenous cephalosporins. She was discharged on oral azithromycin. On follow up after one month, she was asymptomatic.
Figure 1: A computerized tomogram of the abdomen revealed a thickened liver capsule and subcapsular fluid (red arrow), suspicious adhesions between the liver capsule and anterior peritoneum (vertical arrow) and between the inter-bowel loops (arrowheads), diffuse colonic wall thickening and moderate ascites.

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Figure 2: The flimsy “violin string-like adhesions” on laparoscopic examination (arrows).

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    Figures

  [Figure 1], [Figure 2]


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