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CORRESPONDENCE
Year : 2015  |  Volume : 141  |  Issue : 1  |  Page : 128

Authors' response


1 Food and Drug Toxicology Research Centre, National Institute of Nutrition (ICMR), Hyderabad 500 007, Telangana, India
2 Department of Clinical Studies, National Institute of Nutrition (ICMR), Hyderabad 500 007, Telangana, India
3 National Institute of Nutrition (ICMR), Hyderabad 500 007, Telangana, India

Date of Web Publication2-Apr-2015

Correspondence Address:
A L Khandare
Food and Drug Toxicology Research Centre, National Institute of Nutrition (ICMR), Hyderabad 500 007, Telangana
India
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Source of Support: None, Conflict of Interest: None


DOI: 10.4103/0971-5916.154520

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How to cite this article:
Khandare A L, Babu J J, Ankulu M, Aparna N, Shirfule A, Rao G. Authors' response. Indian J Med Res 2015;141:128

How to cite this URL:
Khandare A L, Babu J J, Ankulu M, Aparna N, Shirfule A, Rao G. Authors' response. Indian J Med Res [serial online] 2015 [cited 2019 Oct 20];141:128. Available from: http://www.ijmr.org.in/text.asp?2015/141/1/128/154520

Sir,

Thank you for your appreciation of our efforts made in the study on grass pea consumption [1] . This was a pilot study to know the present scenario of neurolathyrism where the production and consumption were comparatively more. For this purpose we have selected a few blocks, villages of Gondia district randomly based on secondary data available with concerned district authorities. In the study, data were collected on consumption of grass pea, as well as cases of neurolathyrism in the served households (HHs) along with nutritional status. Grass pea samples were collected and ß-ODAP level was estimated which was found considerably lower than earlier reported values.

The consumption of grass pea in the studied population was low and most of the subjects consumed it in the form of gravy. As per an earlier report [2] one needs to consume 300-400 g grass pea (as whole food) continuously for three to four months to get affected, which was missing in the present observation. Since there was less consumption of grass pea, the exposure to ß-ODAP was also less. This could be the reason why we did not find new cases of neurolathyrism in the study villages. As mentioned rightly [3] we wanted to know whether there were any cases of neurolathyrism in the surved villages except in randomly selected HHs. We used this snowball sampling method to locate neurolathyrism cases in the selected villages. By this method we could find two old cases.

This was a pilot study and based on these results a larger study has to be initiated in Chhattisgarh State. The results of the future study will give better idea about grass pea consumption pattern and neurolathyrism.

 
   References Top

1.
Khandare AL, Babu JJ, Ankulu M, Aparna N, Shirfule A, Rao GS. Grass pea consumption & present scenario of neurolathyrism in Maharashtra State of India. Indian J Med Res 2014; 140 : 96-101.  Back to cited text no. 1
    
2.
Ludolph AC, Spencer PS. Toxic models of upper motor neuron disease. J Neurol Sci 1996; 139 (Suppl) : 53-9.  Back to cited text no. 2
    
3.
Raina SK. Establishing association. Indian J Med Res 2015; 141 : 127-8.  Back to cited text no. 3
    




 

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